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Lanosterol induces mitochondrial uncoupling and protects dopaminergic neurons from corpuscle afterlife in a archetypal for Parkinson's disease

  Parkinson's ache (PD) is a neurodegenerative ataxia apparent by the careful decline of dopaminergic neurons in the nigrostriatal pathway. Several curve of affirmation announce that mitochondrial dysfunction contributes to its etiology. Other studies accept appropriate that alterations in sterol homeostasis associate with added accident for PD. Whether these observations are functionally accompanying is, however, unknown.